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Disease Info Card
Anterior Uveitis
Information about Anterior Uveitis: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Anterior Uveitis
Most recent studies have shown that Anterior Uveitis shares some biological mechanisms with ankylosing-spondylitis, aqueous-humor-disorders, arthritis, arthritis-juvenile-rheumatoid, cataract, corneal-diseases, disorder-of-eye, edema, glaucoma, inflammation, intraocular-pressure-disorder, iridocyclitis, keratitis, panuveitis, rheumatoid-arthritis, sarcoidosis, spondylitis, uveitis, uveitis-posterior.
Among the many pathways, these few ones have gauged particular interests from scientists studying Anterior Uveitis, and have been seen in publications frequently: Cell Adhesion, Chemotaxis, Coagulation, Complement Activation, Cytokine Production, Dehiscence, Enucleation, Humoral Immune Response, Hypersensitivity, Immune Response, Inflammatory Response, Leukocyte Migration, Localization, Pathogenesis, Pigmentation, Reflex, Secretion, Sensitization, Viral Replication, Wound Healing
Quite a number of genes have been found to play important roles in Anterior Uveitis, such as ALB, BRCA1, C3, CAT, CCL2, CD4, CTLA4, HLA-B, HLA-DQA1, HLA-DRB4, ICAM1, IL10, IL2, IL6, MBP, MRAP, NOD2, NOS2, TNF. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.