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Disease Info Card
Cortical Dysplasia
Information about Cortical Dysplasia: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Cortical Dysplasia
Most recent studies have shown that Cortical Dysplasia shares some biological mechanisms with brain-diseases, brain-neoplasms, congenital-neurologic-anomalies, dysplasia, ectopic-tissue, epilepsies-partial, epilepsy, epilepsy-temporal-lobe, ganglioglioma, intractable-epilepsy, malformations-of-cortical-development, neoplasms, partial-seizure, polymicrogyria, sclerosis, tuberous-sclerosis.
Among the many pathways, these few ones have gauged particular interests from scientists studying Cortical Dysplasia, and have been seen in publications frequently: Brain Development, Cell Adhesion, Cell Death, Cell Growth, Cell Migration, Cell Proliferation, Cognition, Drug Resistance, Inflammatory Response, Localization, Myelination, Neuroblast Migration, Neuroblast Proliferation, Neurogenesis, Pathogenesis, Programmed Cell Death, Reflex, Segmentation, Synaptic Transmission, Transport
Quite a number of genes have been found to play important roles in Cortical Dysplasia, such as ACAT1, CALB1, CALB2, CD34, DCX, FKTN, GFAP, MAP2, METAP2, MLYCD, MTOR, NES, PVALB, SLC12A3, SLC25A5, TESC, TSC1, TSC2, VIM. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.