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Disease Info Card
Hyponatraemic
Information about Hyponatraemic: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Hyponatraemic
Most recent studies have shown that Hyponatraemic shares some biological mechanisms with brain-diseases, brain-edema, chronic-hyponatremia, comatose, dehydration, demyelination, edema, encephalopathies, heart-failure, hemorrhage, hypernatremia, hypertensive-disease, hyponatraemic-encephalopathy, inappropriate-adh-syndrome, liver-cirrhosis, myelinolysis-central-pontine, vomiting, water-electrolyte-imbalance, water-intoxication.
Among the many pathways, these few ones have gauged particular interests from scientists studying Hyponatraemic, and have been seen in publications frequently: Aging, Aldosterone Secretion, Cell Death, Coagulation, Complement Activation, Diuresis, Excretion, Glomerular Filtration, Hormone Secretion, Ion Transport, Natriuresis, Oxytocin Secretion, Pathogenesis, Response To Water Deprivation, Secretion, Transport, Vasoconstriction, Vasodilation, Vasopressin Secretion, Water Homeostasis
Quite a number of genes have been found to play important roles in Hyponatraemic, such as ACE, AGT, ALB, APCDD1, AQP2, AQP4, AVP, AVPR2, CFTR, CPM, IFNAR1, LAMC2, NLRP3, NPPA, OXT, POMC, REN. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.