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Disease Info Card
Myelinolysis, Central Pontine
Information about Myelinolysis, Central Pontine: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Myelinolysis, Central Pontine
Most recent studies have shown that Myelinolysis, Central Pontine shares some biological mechanisms with brain-diseases, chronic-hyponatremia, comatose, demyelinating-diseases, demyelination, edema, encephalopathies, extrapontine-myelinolysis, hemorrhage, hypernatremia, hyponatraemic, malnutrition, nervous-system-disorder, nervousness, quadriparesis, quadriplegia, wernicke-encephalopathy.
Among the many pathways, these few ones have gauged particular interests from scientists studying Myelinolysis, Central Pontine, and have been seen in publications frequently: Anaphylaxis, Cell Cycle, Cell Death, Cell Division, Coagulation, Cognition, Diuresis, Eating Behavior, Excretion, Hormone Secretion, Localization, Mitosis, Natriuresis, Pathogenesis, Reflex, Secretion, Synaptic Transmission, Transport, Urea Cycle, Vasopressin Secretion
Quite a number of genes have been found to play important roles in Myelinolysis, Central Pontine, such as ACAT1, ATP7B, AVP, CALB2, CD55, CPM, CSF2, FOXM1, GLI3, INS, ISYNA1, LAMC2, MAG, MBP, NOD2, NOS2, SLC25A5, SLC4A1, TRH. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.