pathway Info Card

Complement Activation

Information about Complement Activation: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.

Overview of Complement Activation

Most recent studies have shown that Complement Activation shares some biological mechanisms with blood-coagulation, cell-activation, cell-death, chemotaxis, coagulation, cytokine-production, excretion, fibrinolysis, hypersensitivity, immune-response, inflammatory-response, localization, neutrophil-activation, opsonization, pathogenesis, phagocytosis, platelet-activation, regulation-of-complement-activation, secretion, virulence.

Among the many pathways, these few ones have gauged particular interests from scientists studying Complement Activation, and have been seen in publications frequently: blood-coagulation, cell-activation, cell-death, chemotaxis, coagulation, cytokine-production, excretion, fibrinolysis, hypersensitivity, immune-response, inflammatory-response, localization, neutrophil-activation, opsonization, pathogenesis, phagocytosis, platelet-activation, regulation-of-complement-activation, secretion, virulence

Quite a number of genes have been found to play important roles in Complement Activation, such as ALB, C2, C3, C4A, C5, C5AR1, CD46, CD55, CD59, CFB, CFH, CR1, CRP, HNRNPC, IL6, MBL2, TNF. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.

In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this pathway. Plesae stay updated.