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Disease Info Card
Hypokalemic Periodic Paralysis
Information about Hypokalemic Periodic Paralysis: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Hypokalemic Periodic Paralysis
Most recent studies have shown that Hypokalemic Periodic Paralysis shares some biological mechanisms with acidosis, cardiac-arrhythmia, channelopathies, familial-periodic-paralysis, graves-disease, hereditary-pyropoikilocytosis, hyperkalemic-periodic-paralysis, hyperthyroidism, malignant-hyperpyrexia-due-to-anesthesia, malignant-paraganglionic-neoplasm, muscle-weakness, myopathy, paralysis-flaccid, periodic-paralysis-(finding), quadriplegia, renal-tubular-acidosis, renal-tubular-acidosis-type-i, thyrotoxic-periodic-paralysis, thyrotoxicosis, weakness.
Among the many pathways, these few ones have gauged particular interests from scientists studying Hypokalemic Periodic Paralysis, and have been seen in publications frequently: Aging, Amelogenesis, Cation Transport, Cell Death, Cell Differentiation, Cell Motility, Excretion, Fertilization, Insulin Secretion, Ion Transport, Lipid Storage, Localization, Membrane Depolarization, Muscle Contraction, Pathogenesis, Phagocytosis, Reflex, Secretion, Transepithelial Transport, Transport
Quite a number of genes have been found to play important roles in Hypokalemic Periodic Paralysis, such as ACE, AGT, CACNA1S, CLCN1, DBNL, DPYD, DPYS, INS, KCNE3, KCNJ2, PSMC1, REN, RPS4X, RPS4Y1, RYR1, SCN4A, SLC12A3, SPTA1. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.