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Disease Info Card
Hyperkalemic Periodic Paralysis
Information about Hyperkalemic Periodic Paralysis: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Hyperkalemic Periodic Paralysis
Most recent studies have shown that Hyperkalemic Periodic Paralysis shares some biological mechanisms with cardiac-arrhythmia, channelopathies, dystrophy, familial-periodic-paralysis, hereditary-diseases, hereditary-pyropoikilocytosis, horse-diseases, hypokalemic-periodic-paralysis, malignant-hyperpyrexia-due-to-anesthesia, malignant-paraganglionic-neoplasm, muscle-weakness, myopathy, myotonia-congenita, myotonic-disorders, myotonic-dystrophy, paramyotonia-congenita-(disorder), periodic-paralysis-(finding), weakness.
Among the many pathways, these few ones have gauged particular interests from scientists studying Hyperkalemic Periodic Paralysis, and have been seen in publications frequently: Aging, Cell Adhesion, Cell Migration, Cortisol Secretion, Excretion, Glomerular Filtration, Glycolysis, Ion Transport, Localization, Mating, Membrane Depolarization, Membrane Hyperpolarization, Muscle Contraction, Pathogenesis, Pinocytosis, Reflex, Regulation Of Cell Migration, Regulation Of Ion Transport, Reverse Transcription, Transport
Quite a number of genes have been found to play important roles in Hyperkalemic Periodic Paralysis, such as CACNA1S, CLCN1, CNBP, CST7, DMPK, GH1, INS, KCNE3, KCNJ2, MYCBP2, NPRL3, PAM, POMC, PSMC1, PSMD3, RPS4Y1, SCN4A, SPTA1. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.