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Disease Info Card
Renal Tubular Acidosis
Information about Renal Tubular Acidosis: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Renal Tubular Acidosis
Most recent studies have shown that Renal Tubular Acidosis shares some biological mechanisms with acidosis, diabetes-mellitus, fanconi-syndrome, hypercalciuria, kidney-calculi, kidney-diseases, kidney-failure, kidney-failure-chronic, malignant-neoplasms, metabolic-acidosis, neoplasms, nephrocalcinosis, nephrolithiasis, osteomalacia, proximal-renal-tubular-acidosis, renal-tubular-acidosis-type-i, rickets, sjogrens-syndrome.
Among the many pathways, these few ones have gauged particular interests from scientists studying Renal Tubular Acidosis, and have been seen in publications frequently: Acid Secretion, Anion Transport, Bone Resorption, Cell Cycle, Cell Death, Cell Growth, Cell Proliferation, Depurination, Diuresis, Dna Replication, Endocytosis, Excretion, Glomerular Filtration, Localization, Pathogenesis, Secretion, Translation, Transport, Viral Latency, Viral Replication
Quite a number of genes have been found to play important roles in Renal Tubular Acidosis, such as ATP6V0A4, ATP6V1B1, BBS9, C2, CA2, HOXB2, JUN, KRT8, MRGPRF, MS4A1, PTH, PTRH1, RAPGEF5, RBFOX2, REN, SLC4A1, SLC4A4. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.