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Disease Info Card
Metaphyseal Chondrodysplasia
Information about Metaphyseal Chondrodysplasia: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Metaphyseal Chondrodysplasia
Most recent studies have shown that Metaphyseal Chondrodysplasia shares some biological mechanisms with bone-diseases, bone-diseases-developmental, cartilage-diseases, cartilage-hair-hypoplasia, dwarfism, dysostoses, dysplasia, exocrine-pancreatic-insufficiency, hereditary-multiple-exostoses, hypercalcemia, hypertrophy, hypoplasia, immunologic-deficiency-syndromes, malignant-neoplasms, osteochondrodysplasias, pancreatic-diseases, pancreatic-insufficiency, rickets, shwachman-syndrome.
Among the many pathways, these few ones have gauged particular interests from scientists studying Metaphyseal Chondrodysplasia, and have been seen in publications frequently: Bone Development, Bone Mineralization, Cell Cycle, Cell Death, Cell Proliferation, Chemotaxis, Chondrocyte Differentiation, Chondrocyte Hypertrophy, Chondrocyte Proliferation, Endochondral Ossification, Excretion, Ion Homeostasis, Mitochondrial Rna Processing, Ossification, Pathogenesis, Programmed Cell Death, Rna Processing, Rna Surveillance, Secretion, Translation
Quite a number of genes have been found to play important roles in Metaphyseal Chondrodysplasia, such as COL10A1, FGF23, HHIP, MLYCD, PTH, PTH1R, PTHLH, PTRH1, REG3A, RPL29, SBDS, ST13. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.