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Disease Info Card
Thyroid Associated Opthalmopathies
Information about Thyroid Associated Opthalmopathies: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Thyroid Associated Opthalmopathies
Most recent studies have shown that Thyroid Associated Opthalmopathies shares some biological mechanisms with autoimmune-diseases, autoimmune-reaction, diplopia, disorder-of-eye, disorder-of-the-optic-nerve, edema, exophthalmos, eyelid-retraction, goiter, graves-disease, hashimoto-disease, hyperthyroidism, hypothyroidism, inflammation, intraocular-pressure-disorder, orbit-(eye-disorders), strabismus, thyroid-diseases, thyroiditis.
Among the many pathways, these few ones have gauged particular interests from scientists studying Thyroid Associated Opthalmopathies, and have been seen in publications frequently: Cell Activation, Cell Adhesion, Cell Growth, Cell Proliferation, Cytokine Production, Excretion, Fibroblast Proliferation, Immune Response, Inflammatory Response, Innervation, Localization, Lymphocyte Proliferation, Muscle Hypertrophy, Pathogenesis, Reflex, Secretion, Sensitization, T Cell Activation, Tissue Remodeling, Transposition
Quite a number of genes have been found to play important roles in Thyroid Associated Opthalmopathies, such as BCAR1, CSE1L, CTLA4, CTNND1, GORAB, ICAM1, IFNG, IL1RN, IL2, IL6, PAEP, PPARG, SST, TG, TNF, TPO, TRH, TSHR. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.