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Disease Info Card
Eyelid Retraction
Information about Eyelid Retraction: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Eyelid Retraction
Most recent studies have shown that Eyelid Retraction shares some biological mechanisms with blepharoptosis, cicatrix, conjunctival-diseases, diplopia, disorder-of-eye, ectropion, edema, entropion, exophthalmos, eyelid-diseases, graves-disease, hyperthyroidism, inflammation, lagophthalmos, strabismus, thyroid-associated-opthalmopathies, thyroid-diseases.
Among the many pathways, these few ones have gauged particular interests from scientists studying Eyelid Retraction, and have been seen in publications frequently: Aging, Dehiscence, Enucleation, Fibroblast Migration, Hemostasis, Inflammatory Response, Innervation, Localization, Muscle Atrophy, Muscle Contraction, Muscle Hypertrophy, Optic Vesicle Formation, Pathogenesis, Reflex, Regeneration, Secretion, Sensitization, Striated Muscle Atrophy, Swimming, Transposition
Quite a number of genes have been found to play important roles in Eyelid Retraction, such as ALOX5AP, ATXN1, ATXN2, ATXN3, BCAR1, CASQ1, CSE1L, CTNND1, GORAB, MBD5, PLXNA2, PSPH, SLC25A5, STXBP3, TRH, TSHR. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.