Disease Info Card

Thin Basement Membrane Disease

Information about Thin Basement Membrane Disease: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.

Overview of Thin Basement Membrane Disease

Most recent studies have shown that Thin Basement Membrane Disease shares some biological mechanisms with alport-syndrome-x-linked, familial-hematuria, focal-glomerulosclerosis, glioblastoma, glomerulonephritis, glomerulonephritis-minimal-change, glomerulosclerosis-(disorder), hereditary-nephritis, hypertensive-disease, iga-glomerulonephritis, kidney-diseases, kidney-failure, kidney-failure-chronic, membranous-glomerulonephritis, microscopic-hematuria, nephritis, nephrotic-syndrome, proteinuria-of-undiagnosed-cause, renal-glomerular-disease, segmental-glomerulosclerosis.

Among the many pathways, these few ones have gauged particular interests from scientists studying Thin Basement Membrane Disease, and have been seen in publications frequently: Aging, Cell Activation, Coagulation, Complement Activation, Excretion, Glomerular Filtration, Intracellular Signal Transduction, Methylation, Pathogenesis, Pigmentation, Reflex, Transposition

Quite a number of genes have been found to play important roles in Thin Basement Membrane Disease, such as ACTN4, ALB, C3, CALB2, CD55, CFHR5, COL4A3, COL4A4, COL4A5, CR2, CTLA4, DHRS4, GNL3, HLA-DQA1, MLYCD, NOD2, UMOD. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.

In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.