Disease Info Card

Microscopic Hematuria

Information about Microscopic Hematuria: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.

Overview of Microscopic Hematuria

Most recent studies have shown that Microscopic Hematuria shares some biological mechanisms with bladder-neoplasm, carcinoma, edema, glomerulonephritis, hemorrhage, hypertensive-disease, iga-glomerulonephritis, kidney-diseases, kidney-failure, kidney-failure-chronic, malignant-neoplasm-of-urinary-bladder, malignant-neoplasms, neoplasms, nephritis, nephrotic-syndrome, pain, proteinuria-of-undiagnosed-cause, renal-glomerular-disease, urinary-tract-infection, vasculitis.

Among the many pathways, these few ones have gauged particular interests from scientists studying Microscopic Hematuria, and have been seen in publications frequently: Cell Proliferation, Coagulation, Complement Activation, Diuresis, Donor Selection, Enucleation, Excretion, Flight, Glomerular Filtration, Glycosylation, Hypersensitivity, Immune Response, Inflammatory Response, Localization, Micturition, Pathogenesis, Platelet Aggregation, Secretion, Sensitization, Transport

Quite a number of genes have been found to play important roles in Microscopic Hematuria, such as ACE, ACTN4, ALB, C3, C4A, CALB2, CD55, CFHR5, CR2, CRP, DHRS4, FN1, KLK3, LYZ, MPO, RAPGEF5, TDGF1, TNF. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.

In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.