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- Table of Contents
Information about Polyglandular Type I Autoimmune Syndrome: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Most recent studies have shown that Polyglandular Type I Autoimmune Syndrome shares some biological mechanisms with addison-disease, adrenal-gland-hypofunction, autoimmune-diseases, autoimmune-polyendocrinopathy-syndrome-type-1, autoimmune-reaction, autoimmunity, candidiasis, candidiasis-chronic-mucocutaneous, diabetes-mellitus, diabetes-mellitus-insulin-dependent, dystrophy, endocrine-system-diseases, hepatitis, hypoparathyroidism, hypothyroidism, immunologic-tolerance, oral-candidiasis, polyendocrinopathies-autoimmune.
Among the many pathways, these few ones have gauged particular interests from scientists studying Polyglandular Type I Autoimmune Syndrome, and have been seen in publications frequently: Cell Development, Cell Maturation, Cell Proliferation, Cytokine Production, Developmental Process, Hypersensitivity, Immune Response, Localization, Methylation, Nuclear Transport, Ossification, Pathogenesis, Pigmentation, Regulation Of Gene Expression, Secretion, T Cell Selection, Tolerance Induction, Translation, Transport, Virulence
Quite a number of genes have been found to play important roles in Polyglandular Type I Autoimmune Syndrome, such as AIRE, CARD9, CD4, CD8A, CREBBP, CTLA4, CYP11A1, CYP17A1, CYP21A2, FOXP3, HLA-DRB4, IL22, INS, KLK3, NUDT11, POMC, PTH, PTRH1, SH2B2. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.