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Disease Info Card
Pendular Nystagmus
Information about Pendular Nystagmus: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Pendular Nystagmus
Most recent studies have shown that Pendular Nystagmus shares some biological mechanisms with albinism, ataxia, atrophy, brain-diseases, congenital-nystagmus, hypoplasia, jerk-nystagmus, movement-disorders, multiple-sclerosis, myoclonus, nystagmus, ocular-motility-disorders, ophthalmoplegia, periodic-alternating-nystagmus, sclerosis, strabismus, visual-impairment.
Among the many pathways, these few ones have gauged particular interests from scientists studying Pendular Nystagmus, and have been seen in publications frequently: Dehiscence, Excretion, Immune Response, Innervation, Muscle Atrophy, Myelination, Pathogenesis, Pigmentation, Proprioception, Reflex, Response To Biotin, Synaptic Transmission, Visual Behavior
Quite a number of genes have been found to play important roles in Pendular Nystagmus, such as ANPEP, CACNA1A, CGN, CSF2, DCX, ERG, FRMD7, GFAP, KCNH2, LAMC2, MANBA, MAPT, OPTC, PDXP, PLP1, ROBO3, RPE65, TYR. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.
Pathways Related to Pendular Nystagmus
This information is being compiled and will come in a future update
| Dehiscence | Excretion | Immune Response |
| Innervation | Muscle Atrophy | Myelination |
| Pathogenesis | Pigmentation | Proprioception |
| Reflex | Response To Biotin | Synaptic Transmission |
| Visual Behavior |