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Disease Info Card
Ocular Motility Disorders
Information about Ocular Motility Disorders: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Ocular Motility Disorders
Most recent studies have shown that Ocular Motility Disorders shares some biological mechanisms with ataxia, atrophy, blepharoptosis, brown-tendon-sheath-syndrome, diplopia, fracture, internuclear-ophthalmoplegia, malnutrition, movement-disorders, multiple-sclerosis, myoclonus, neoplasms, nervousness, nystagmus, ophthalmoplegia, orbital-fractures, schizophrenia, sclerosis, strabismus.
Among the many pathways, these few ones have gauged particular interests from scientists studying Ocular Motility Disorders, and have been seen in publications frequently: Aging, Axon Guidance, Brainstem Development, Cognition, Dna Repair, Flight, Hypersensitivity, Immune Response, Innervation, Localization, Muscle Atrophy, Muscle Contraction, Myelination, Pathogenesis, Proprioception, Reflex, Regeneration, Secretion, Transposition, Visual Perception
Quite a number of genes have been found to play important roles in Ocular Motility Disorders, such as AFP, APTX, CACNA1A, CRLF1, CSF2, DTL, HOXA1, KIF21A, LAMC2, MSMB, PAMR1, PHOX2A, POMC, RANGAP1, ROBO3, SETX, SLC17A5, ZMYM2. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.