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Disease Info Card
Mutation, Out-of-frame
Information about Mutation, Out-of-frame: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Mutation, Out-of-frame
Most recent studies have shown that Mutation, Out-of-frame shares some biological mechanisms with adenocarcinoma, carcinogenesis, carcinoma, cell-transformation-neoplastic, colorectal-cancer, colorectal-neoplasms, hereditary-nonpolyposis-colorectal-cancer, high-frequency-microsatellite-instability, malignant-neoplasm-of-breast, malignant-neoplasm-of-stomach, malignant-neoplasms, malignant-tumor-of-colon, mammary-neoplasms, microsatellite-instability, neoplasms, polyposis, salmonella-infections, stomach-neoplasms.
Among the many pathways, these few ones have gauged particular interests from scientists studying Mutation, Out-of-frame, and have been seen in publications frequently: Cell Cycle, Cell Death, Cell Growth, Cell Proliferation, Dna Repair, Dna Replication, Drug Resistance, Gene Conversion, Localization, Methylation, Mismatch Repair, Pathogenesis, Recombinational Repair, Reverse Transcription, Rna Splicing, Secretion, Translation, Translesion Synthesis, Transport, Virulence
Quite a number of genes have been found to play important roles in Mutation, Out-of-frame, such as APC, BAX, BRCA1, BRCA2, CFTR, DMD, GLB1, HPRT1, MECP2, MLH1, MRC1, MSH2, MSH3, MSH6, PROC, TCF7L2, TGFBR2, TP53. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.