Disease Info Card

Interfrontal Craniofaciosynostosis

Information about Interfrontal Craniofaciosynostosis: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.

Overview of Interfrontal Craniofaciosynostosis

Most recent studies have shown that Interfrontal Craniofaciosynostosis shares some biological mechanisms with blood-loss-surgical, congenital-abnormal-synostosis, congenital-abnormality, congenital-heart-defects, craniosynostosis, cytogenetic-abnormality, developmental-delay-(disorder), developmental-disabilities, epilepsy, haploinsufficiency, hemorrhage, hydrocephalus, interparietal-craniosynostosis, microcephaly, pregnancy-complications, scaphycephaly, trigonocephaly, trigonocephaly-syndrome.

Among the many pathways, these few ones have gauged particular interests from scientists studying Interfrontal Craniofaciosynostosis, and have been seen in publications frequently: Cell Growth, Inflammatory Response, Osteoblast Differentiation, Wound Healing

Quite a number of genes have been found to play important roles in Interfrontal Craniofaciosynostosis, such as BMP2, CDKN2A, CDON, CER1, FBN2, FGFR1, FGFR2, FREM1, FUT2, GLI3, MB, NF1, PTCH1, RFC1, RFC2, RUNX2, TINAGL1, TWIST1. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.

In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.

Interfrontal Craniofaciosynostosis Related Genes

click to see detail information for each gene

BMP2 CDKN2A CDON
CER1 FBN2 FGFR1
FGFR2 FREM1 FUT2
GLI3 MB NF1
PTCH1 RFC1 RFC2
RUNX2 TINAGL1 TWIST1

Pathways Related to Interfrontal Craniofaciosynostosis

This information is being compiled and will come in a future update

Cell Growth Inflammatory Response Osteoblast Differentiation
Wound Healing