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Disease Info Card
Giant Cell Arteritis
Information about Giant Cell Arteritis: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Giant Cell Arteritis
Most recent studies have shown that Giant Cell Arteritis shares some biological mechanisms with arteritis, arthritis, blind-vision, claudication-(finding), disorder-of-the-optic-nerve, headache, inflammation, ischemia, optic-neuropathy-ischemic, pain, polyarteritis-nodosa, polymyalgia-rheumatica, rheumatoid-arthritis, takayasu-arteritis, temporal-arteritis, vasculitis, visual-impairment, wegener-granulomatosis.
Among the many pathways, these few ones have gauged particular interests from scientists studying Giant Cell Arteritis, and have been seen in publications frequently: Acute-phase Response, Aging, Angiogenesis, Cell Activation, Cell Adhesion, Coagulation, Cytokine Production, Granuloma Formation, Hypersensitivity, Immune Response, Inflammatory Response, Localization, Macrophage Activation, Mastication, Menopause, Pathogenesis, Platelet Aggregation, Secretion, T Cell Activation, Tropism
Quite a number of genes have been found to play important roles in Giant Cell Arteritis, such as ACR, C3, CD4, CRP, CSRP1, DDX4, ELN, ESR1, GCA, HLA-DQA1, HLA-DRB1, HLA-DRB3, HLA-DRB4, HLA-DRB5, IL2, IL6, NOD2, TLR4, TNF, TNFRSF10A. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.