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Disease Info Card
Wegener Granulomatosis
Information about Wegener Granulomatosis: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Wegener Granulomatosis
Most recent studies have shown that Wegener Granulomatosis shares some biological mechanisms with arteritis, arthritis, autoimmune-diseases, autoimmune-reaction, churg-strauss-syndrome, glomerulonephritis, granuloma, hemorrhage, inflammation, kidney-diseases, kidney-failure, lung-diseases, lupus-erythematosus-systemic, microscopic-polyangiitis, polyarteritis-nodosa, rheumatoid-arthritis, sarcoidosis, vasculitis.
Among the many pathways, these few ones have gauged particular interests from scientists studying Wegener Granulomatosis, and have been seen in publications frequently: Angiogenesis, Cell Activation, Cell Adhesion, Cell Proliferation, Chemotaxis, Coagulation, Cytokine Production, Glomerular Filtration, Glycosylation, Granuloma Formation, Hypersensitivity, Immune Response, Inflammatory Response, Localization, Neutrophil Activation, Pathogenesis, Phagocytosis, Respiratory Burst, Secretion, T Cell Activation
Quite a number of genes have been found to play important roles in Wegener Granulomatosis, such as ACR, CD4, CMAS, CRP, CSRP1, CTLA4, CTSG, CYCS, ELANE, ESR1, GYPA, HLA-DQA1, IL2, IL6, MPO, NOD2, PRTN3, TNF. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.