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Disease Info Card
Temporal Arteritis
Information about Temporal Arteritis: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Temporal Arteritis
Most recent studies have shown that Temporal Arteritis shares some biological mechanisms with arteritis, arthritis, blind-vision, claudication-(finding), giant-cell-arteritis, headache, inflammation, ischemia, neoplasms, pain, polyarteritis-nodosa, polymyalgia-rheumatica, rheumatism, rheumatoid-arthritis, stenosis, takayasu-arteritis, vasculitis, visual-impairment.
Among the many pathways, these few ones have gauged particular interests from scientists studying Temporal Arteritis, and have been seen in publications frequently: Aging, Cell Adhesion, Coagulation, Granuloma Formation, Hypersensitivity, Immune Response, Inflammatory Response, Localization, Mastication, Muscle Contraction, Pathogenesis, Platelet Aggregation, Reflex, Secretion, Senescence, Sensitization, Transport, Tropism, Vasoconstriction, Virulence
Quite a number of genes have been found to play important roles in Temporal Arteritis, such as ACR, ALB, BEST1, C3, CRP, CSRP1, DDX4, DMD, ELANE, ELN, EMD, ESR1, GCA, HLA-DRB4, HP, IL2, IL6, POMC, TNF, TNFRSF10A. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.