Disease Info Card

Anemia, Pernicious

Information about Anemia, Pernicious: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.

Overview of Anemia, Pernicious

Most recent studies have shown that Anemia, Pernicious shares some biological mechanisms with achlorhydria, anemia, anemia-macrocytic, anemia-megaloblastic, atrophy, autoimmune-diseases, autoimmune-reaction, carcinoma, diabetes-mellitus, folic-acid-deficiency, gastritis, gastritis-atrophic, hypochromic-anemia, malabsorption-syndrome, malignant-neoplasm-of-stomach, malignant-neoplasms, neoplasms, stomach-neoplasms, ulcer, vitamin-b-12-deficiency.

Among the many pathways, these few ones have gauged particular interests from scientists studying Anemia, Pernicious, and have been seen in publications frequently: Acid Secretion, Aging, Cell Proliferation, Coagulation, Excretion, Gastric Acid Secretion, Gastric Emptying, Hemopoiesis, Hypersensitivity, Immune Response, Intestinal Absorption, Localization, Methylation, Pathogenesis, Phagocytosis, Pigmentation, Regeneration, Response To Vitamin, Secretion, Transport

Quite a number of genes have been found to play important roles in Anemia, Pernicious, such as AMY2A, BLOC1S6, CCL21, EPB42, FLVCR1, GAST, INS, PGA3, PGA4, PGA5, PRH1, SCT, SST, TCN2, TF, TG, TPO. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.

In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.