This website uses cookies to ensure you get the best experience on our website.
- Table of Contents
, No Gclids
Disease Info Card
Intraocular Lymphoma
Information about Intraocular Lymphoma: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Intraocular Lymphoma
Most recent studies have shown that Intraocular Lymphoma shares some biological mechanisms with aphakia, capsule-opacification, cataract, corneal-diseases, dislocations, edema, endophthalmitis, glaucoma, inflammation, intraocular-pressure-disorder, intraoperative-complications, lymphoma, macular-retinal-edema, myopia, posterior-capsule-opacification, refractive-errors, retinal-detachment, scleral-diseases, uveitis.
Among the many pathways, these few ones have gauged particular interests from scientists studying Intraocular Lymphoma, and have been seen in publications frequently: Aging, Biofilm Formation, Cell Adhesion, Cell Death, Cell Growth, Cell Migration, Cell Proliferation, Dehiscence, Enucleation, Epithelial Cell Migration, Epithelial Cell Proliferation, Inflammatory Response, Lens Epithelial Cell Proliferation, Localization, Muscle Contraction, Pathogenesis, Reflex, Translation, Transport, Wound Healing
Quite a number of genes have been found to play important roles in Intraocular Lymphoma, such as ACD, ASAH1, BRCA1, BTG3, CALCR, CS, FTMT, FUT2, IL10, IL6, PLK3, RANGAP1, RPS6KB2, SLC17A5, SQLE, TNFSF14, ZAP70. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.