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Disease Info Card
Exertional Rhabdomyolysis
Information about Exertional Rhabdomyolysis: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Exertional Rhabdomyolysis
Most recent studies have shown that Exertional Rhabdomyolysis shares some biological mechanisms with acidosis, acute-kidney-injury, bird-diseases, dehydration, edema, glycogen-storage-disease, horse-diseases, kidney-failure, kidney-failure-acute, malignant-hyperpyrexia-due-to-anesthesia, malignant-paraganglionic-neoplasm, muscle-injury, myalgia, myopathy, pain, physiological-stress, rhabdomyolysis, sickle-cell-trait, weakness.
Among the many pathways, these few ones have gauged particular interests from scientists studying Exertional Rhabdomyolysis, and have been seen in publications frequently: Coagulation, Diuresis, Excretion, Fatty Acid Oxidation, Glomerular Filtration, Glycolysis, Heat Dissipation, Inflammatory Response, Lactate Transport, Localization, Muscle Atrophy, Muscle Contraction, Oxidative Phosphorylation, Pathogenesis, Platelet Activation, Regeneration, Regulation Of Muscle Contraction, Swimming, Transport, Vasopressin Secretion
Quite a number of genes have been found to play important roles in Exertional Rhabdomyolysis, such as ACE, AVP, CACNA1S, CHKA, CHKB, GYS1, IK, IL6, INS, KCNH8, MB, MYLK, MYLK2, MYLK3, MYOD1, PIK3C2A, RYR1, SLC16A1, SLC17A5. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.