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Disease Info Card
Rhabdomyolysis
Information about Rhabdomyolysis: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Rhabdomyolysis
Most recent studies have shown that Rhabdomyolysis shares some biological mechanisms with acidosis, acute-kidney-injury, comatose, compartment-syndromes, edema, exertional-rhabdomyolysis, hypercholesterolemia, hyperlipidemia, kidney-failure, kidney-failure-acute, malignant-hyperpyrexia-due-to-anesthesia, malignant-paraganglionic-neoplasm, muscle-weakness, myalgia, myopathy, myositis, pain, poisoning, weakness.
Among the many pathways, these few ones have gauged particular interests from scientists studying Rhabdomyolysis, and have been seen in publications frequently: Anaphylaxis, Cell Death, Coagulation, Diuresis, Excretion, Fatty Acid Beta-oxidation, Fatty Acid Oxidation, Glomerular Filtration, Glycolysis, Hypersensitivity, Inflammatory Response, Lipid Storage, Localization, Muscle Atrophy, Oxidative Phosphorylation, Pathogenesis, Regeneration, Secretion, Transport, Vasoconstriction
Quite a number of genes have been found to play important roles in Rhabdomyolysis, such as ACADVL, ALB, CAT, CDKN2A, CHKA, CHKB, CHPT1, CPT2, CYP3A4, DMD, HMGCR, INS, MB, PIK3C2A, PTH, RYR1, SLC17A5, TNF. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.