This website uses cookies to ensure you get the best experience on our website.
- Table of Contents
, No Gclids
Disease Info Card
Acute Kidney Injury
Information about Acute Kidney Injury: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Acute Kidney Injury
Most recent studies have shown that Acute Kidney Injury shares some biological mechanisms with acute-kidney-insufficiency, anuria, glomerulonephritis, hemorrhage, hypertensive-disease, injury-to-kidney, ischemia, kidney-diseases, kidney-failure, kidney-failure-acute, kidney-failure-chronic, kidney-tubular-necrosis-acute, nephritis, oliguria, renal-insufficiency, renal-tubular-necrosis, rhabdomyolysis, systemic-infection, uremia.
Among the many pathways, these few ones have gauged particular interests from scientists studying Acute Kidney Injury, and have been seen in publications frequently: Aging, Cell Adhesion, Cell Cycle, Cell Death, Cell Proliferation, Coagulation, Diuresis, Excretion, Glomerular Filtration, Hypersensitivity, Immune Response, Inflammatory Response, Localization, Natriuresis, Pathogenesis, Regeneration, Secretion, Transport, Vasoconstriction, Vasodilation
Quite a number of genes have been found to play important roles in Acute Kidney Injury, such as ACE, AGT, ALB, CALB2, CD55, CDKN2A, CST3, EPO, HAVCR1, IL18, IL6, INS, LCN2, LRSAM1, MB, NOS2, PTH, RAPGEF5, REN, TNF. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.