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- Table of Contents
Information about Epidermolysis Bullosa Dystrophica: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Most recent studies have shown that Epidermolysis Bullosa Dystrophica shares some biological mechanisms with bulla, carcinoma, cicatrix, cockayne-touraine-disease, dermatologic-disorders, dystrophy, epidermolysis-bullosa, esophageal-stenosis, hallopeau-siemens-disease, junctional-epidermolysis-bullosa, malignant-squamous-cell-neoplasm, milium-cyst, mucinous-adenocarcinoma, neoplasms, skin-diseases-genetic, skin-neoplasms, stenosis, tissue-adhesions.
Among the many pathways, these few ones have gauged particular interests from scientists studying Epidermolysis Bullosa Dystrophica, and have been seen in publications frequently: Cell Adhesion, Cell Differentiation, Cell Motility, Fibroblast Proliferation, Heteroduplex Formation, Immune Response, Keratinization, Localization, Mastication, Mating, Pathogenesis, Pigmentation, Regeneration, Secretion, Tooth Eruption, Translation, Transport, Transposition, Wound Healing
Quite a number of genes have been found to play important roles in Epidermolysis Bullosa Dystrophica, such as C7, CCDC6, COL17A1, COL7A1, CTLA4, CXCL10, DST, F9, FLG, FN1, HLA-DQA1, MMP1, MMP7, NOD2, PLEC, PLOD1, PTCH1, RET, SERPINB3, TAS2R38. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.