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Disease Info Card
Hallopeau-siemens Disease
Information about Hallopeau-siemens Disease: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Hallopeau-siemens Disease
Most recent studies have shown that Hallopeau-siemens Disease shares some biological mechanisms with bulla, carcinoma, cicatrix, dermatologic-disorders, epidermolysis-bullosa, epidermolysis-bullosa-dystrophica, esophageal-stenosis, hereditary-diseases, malignant-neoplasms, malignant-squamous-cell-neoplasm, mucinous-adenocarcinoma, neoplasms, pathological-dilatation, skin-diseases-genetic, skin-neoplasms, squamous-cell-carcinoma-of-skin, stenosis, ulcer.
Among the many pathways, these few ones have gauged particular interests from scientists studying Hallopeau-siemens Disease, and have been seen in publications frequently: Cell Adhesion, Cell Differentiation, Fibroblast Proliferation, Hormone Secretion, Hypersensitivity, Immune Response, Keratinization, Keratinocyte Differentiation, Localization, Mating, Pathogenesis, Pigmentation, Proteolysis, Response To Wounding, Secretion, Tooth Eruption, Translation, Transport, Wound Healing
Quite a number of genes have been found to play important roles in Hallopeau-siemens Disease, such as C7, CCDC6, COL7A1, CXCL10, F9, FBN1, ITGA2B, IVL, LYZ, MAPK3, MMP1, MMP7, MMP9, PLOD1, PTCH1, RET, SERPINB3, TAS2R38. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.