Disease Info Card

Chronic Atrial Fibrillation

Information about Chronic Atrial Fibrillation: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.

Overview of Chronic Atrial Fibrillation

Most recent studies have shown that Chronic Atrial Fibrillation shares some biological mechanisms with atrial-fibrillation, atrial-flutter, cardiac-arrhythmia, cardiac-fibrillation, cardiomyopathies, cerebrovascular-accident, diseases-of-mitral-valve, embolism, heart-diseases, heart-failure, heart-valve-disease, hemorrhage, hypertensive-disease, infarction, mitral-valve-insufficiency, mitral-valve-stenosis, paroxysmal-atrial-fibrillation, rheumatism, stenosis, thromboembolism.

Among the many pathways, these few ones have gauged particular interests from scientists studying Chronic Atrial Fibrillation, and have been seen in publications frequently: Aging, Cardiac Conduction, Circadian Rhythm, Coagulation, Dedifferentiation, Excretion, Fibrinolysis, Flight, Glomerular Filtration, Hemostasis, Hibernation, Hyperphosphorylation, Hypersensitivity, Localization, Pathogenesis, Platelet Activation, Platelet Aggregation, Reflex, Secretion, Transport

Quite a number of genes have been found to play important roles in Chronic Atrial Fibrillation, such as ACE, CALCR, CALR, CHAF1A, DCX, F2, GJA1, GJA5, GNL3, KAT2B, KRAS, KRT5, NPPB, PSMD4, SCTR, SLC6A8, SNCG, VWF. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.

In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.