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- Table of Contents
Information about Cerebral Amyloid Angiopathy: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Most recent studies have shown that Cerebral Amyloid Angiopathy shares some biological mechanisms with alzheimers-disease, amyloid-deposition, amyloidosis, cerebral-hemorrhage, cerebrovascular-accident, cerebrovascular-disorders, dementia, dementia-vascular, dental-plaque, hematoma, hemorrhage, hypertensive-disease, impaired-cognition, infarction, infectious-canine-hepatitis, neurofibrillary-tangles, plaque-amyloid, senile-cerebral-amyloid-angiopathy, senile-plaques, vascular-diseases.
Among the many pathways, these few ones have gauged particular interests from scientists studying Cerebral Amyloid Angiopathy, and have been seen in publications frequently: Aging, Amyloid Fibril Formation, Angiogenesis, Cell Death, Cell Proliferation, Cholesterol Transport, Coagulation, Cognition, Hemostasis, Immune Response, Inflammatory Response, Localization, Pathogenesis, Phagocytosis, Proteolysis, Secretion, Senescence, Transport, Tropism, Vasoconstriction
Quite a number of genes have been found to play important roles in Cerebral Amyloid Angiopathy, such as ABCB6, ACE, APOE, APP, ATP6V0A4, COL4A2, CSF2, CST3, ITM2B, LAMC2, MAPT, MME, PLP2, PRDX2, PRNP, PSEN1, SGCG, TSHZ1, TTR. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.