Disease Info Card

Neurofibrillary Tangles

Information about Neurofibrillary Tangles: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.

Overview of Neurofibrillary Tangles

Most recent studies have shown that Neurofibrillary Tangles shares some biological mechanisms with alzheimers-disease, atrophy, cognition-disorders, dementia, dental-plaque, gliosis, impaired-cognition, nerve-degeneration, neurodegenerative-disorders, neuropathology-disease, neuropil-threads, paired-helical-filament, parkinson-disease, plaque-amyloid, progressive-supranuclear-palsy, secondary-parkinson-disease, senile-plaques, supranuclear-palsy, tauopathies, vascular-diseases.

Among the many pathways, these few ones have gauged particular interests from scientists studying Neurofibrillary Tangles, and have been seen in publications frequently: Aging, Autophagy, Cell Cycle, Cell Death, Cognition, Glycosylation, Hyperphosphorylation, Inflammatory Response, Innervation, Localization, Neurogenesis, Neuroprotection, Pathogenesis, Protein Oxidation, Protein Phosphorylation, Protein Processing, Proteolysis, Regeneration, Secretion, Transport

Quite a number of genes have been found to play important roles in Neurofibrillary Tangles, such as ACHE, APOE, APP, BPIFA2, CA1, CDK5, CSF2, GFAP, LAMC2, MAPT, MSMB, PSEN1, PSEN2, PSPH, PSPN, REG1A, SGCG, SNCA, STXBP3. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.

In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.