Disease Info Card

Amyloid Deposition

Information about Amyloid Deposition: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.

Overview of Amyloid Deposition

Most recent studies have shown that Amyloid Deposition shares some biological mechanisms with alzheimers-disease, amyloidosis, cerebral-amyloid-angiopathy, dementia, dental-plaque, diabetes-mellitus, hemorrhage, impaired-cognition, inflammation, kidney-diseases, neoplasms, neurofibrillary-tangles, plaque-amyloid, polyneuropathy, primary-amyloidosis, primary-systemic-amyloidosis, secondary-amyloidosis, senile-cerebral-amyloid-angiopathy, senile-plaques, vascular-diseases.

Among the many pathways, these few ones have gauged particular interests from scientists studying Amyloid Deposition, and have been seen in publications frequently: Aging, Amyloid Fibril Formation, Cell Cycle, Cell Death, Coagulation, Cognition, Glycosylation, Hyperphosphorylation, Immune Response, Inflammatory Response, Innervation, Insulin Secretion, Localization, Neurogenesis, Pathogenesis, Phagocytosis, Proteolysis, Secretion, Senescence, Transport

Quite a number of genes have been found to play important roles in Amyloid Deposition, such as APCS, APOE, APP, ATP6V0A4, CEL, CSF2, CST3, IAPP, INS, LAMC2, LYZ, MAPT, PLP2, PRNP, PSEN1, SGCG, TEAD1, TG, TTR. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.

In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.