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Disease Info Card
Carotid Artery Injuries
Information about Carotid Artery Injuries: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Carotid Artery Injuries
Most recent studies have shown that Carotid Artery Injuries shares some biological mechanisms with aneurysm, arterial-injury, arteriovenous-fistula, carotid-artery-diseases, carotid-artery-thrombosis, craniocerebral-trauma, formation-neointima, hemorrhage, hyperplasia, neck-injuries, neointima, nonpenetrating-wounds, pathologic-fistula, pseudoaneurysm, stenosis, thrombosis, vascular-system-injuries, wounds-penetrating.
Among the many pathways, these few ones have gauged particular interests from scientists studying Carotid Artery Injuries, and have been seen in publications frequently: Angiogenesis, Cell Adhesion, Cell Cycle, Cell Death, Cell Growth, Cell Migration, Cell Proliferation, Coagulation, Hemostasis, Inflammatory Response, Localization, Muscle Cell Migration, Muscle Cell Proliferation, Pathogenesis, Platelet Activation, Platelet Aggregation, Regeneration, Secretion, Smooth Muscle Cell Migration, Smooth Muscle Cell Proliferation
Quite a number of genes have been found to play important roles in Carotid Artery Injuries, such as ACE, AGT, AKT1, CCL2, F3, FGF2, ICA, MAPK1, MAPK3, MMP2, MMP9, NFKB1, NOS2, NOS3, PCNA, PLAU, PLG, TNF, VEGFA. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.