Disease Info Card

Neointima

Information about Neointima: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.

Overview of Neointima

Most recent studies have shown that Neointima shares some biological mechanisms with arterial-injury, arteriosclerosis, atherosclerosis, carotid-artery-injuries, coronary-artery-disease, coronary-heart-disease, coronary-restenosis, dental-plaque, formation-neointima, graft-occlusion-vascular, hyperplasia, inflammation, stenosis, thrombosis, thrombus, tissue-adhesions, vascular-diseases, vascular-system-injuries.

Among the many pathways, these few ones have gauged particular interests from scientists studying Neointima, and have been seen in publications frequently: Angiogenesis, Cell Adhesion, Cell Cycle, Cell Death, Cell Growth, Cell Migration, Cell Proliferation, Chemotaxis, Coagulation, Inflammatory Response, Localization, Muscle Cell Migration, Muscle Cell Proliferation, Pathogenesis, Platelet Aggregation, Regeneration, Secretion, Smooth Muscle Cell Migration, Smooth Muscle Cell Proliferation, Wound Healing

Quite a number of genes have been found to play important roles in Neointima, such as ACE, AGT, CCL2, CDKN1A, ELN, FGF2, IL6, MAPK1, MAPK3, MMP2, MMP9, NOS2, NOS3, PCNA, PLAU, PLG, SPP1, TNF, VEGFA. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.

In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.

Neointima Related Genes

click to see detail information for each gene

ACE AGT CCL2
CDKN1A ELN FGF2
IL6 MAPK1 MAPK3
MMP2 MMP9 NOS2
NOS3 PCNA PLAU
PLG SPP1 TNF
VEGFA