Disease Info Card

Basal Cell Neoplasm

Information about Basal Cell Neoplasm: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.

Overview of Basal Cell Neoplasm

Most recent studies have shown that Basal Cell Neoplasm shares some biological mechanisms with adenocarcinoma, adenoid-cystic-carcinoma, basal-cell-carcinoma, carcinoma, cell-transformation-neoplastic, desmoplastic, epithelioma, facial-neoplasms, malignant-neoplasm-of-breast, malignant-neoplasm-of-skin, malignant-neoplasms, malignant-paraganglionic-neoplasm, malignant-squamous-cell-neoplasm, mammary-neoplasms, melanoma, neoplasm-metastasis, neoplasms, neoplasms-multiple-primary, skin-neoplasms, trichoepithelioma.

Among the many pathways, these few ones have gauged particular interests from scientists studying Basal Cell Neoplasm, and have been seen in publications frequently: Anagen, Catagen, Cell Adhesion, Cell Cycle, Cell Death, Cell Differentiation, Cell Growth, Cell Proliferation, Dna Methylation, Dna Repair, Hair Follicle Development, Immune Response, Keratinization, Localization, Methylation, Pathogenesis, Pigmentation, Programmed Cell Death, Telogen

Quite a number of genes have been found to play important roles in Basal Cell Neoplasm, such as BCL2, BRCA1, CD34, CHKA, CHKB, CKAP4, CYLD, EGFR, ERBB2, ESR1, KRT14, KRT20, KRT5, PGR, RPE65, TP53, TP63, UVRAG, VIM. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.

In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.

Basal Cell Neoplasm Related Genes

click to see detail information for each gene

BCL2 BRCA1 CD34
CHKA CHKB CKAP4
CYLD EGFR ERBB2
ESR1 KRT14 KRT20
KRT5 PGR RPE65
TP53 TP63 UVRAG
VIM