Disease Info Card

Neoplasms, Multiple Primary

Information about Neoplasms, Multiple Primary: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.

Overview of Neoplasms, Multiple Primary

Most recent studies have shown that Neoplasms, Multiple Primary shares some biological mechanisms with adenocarcinoma, adenoma, carcinoma, colonic-neoplasms, kidney-neoplasm, liver-neoplasms, lung-neoplasms, lymphoma, malignant-neoplasm-of-breast, malignant-neoplasms, malignant-paraganglionic-neoplasm, malignant-squamous-cell-neoplasm, mammary-neoplasms, melanoma, neoplasm-metastasis, neoplasm-recurrence-local, neoplasms, ovarian-neoplasm, skin-neoplasms, stomach-neoplasms.

Among the many pathways, these few ones have gauged particular interests from scientists studying Neoplasms, Multiple Primary, and have been seen in publications frequently: Aging, Cell Cycle, Cell Differentiation, Cell Proliferation, Coagulation, Dedifferentiation, Dna Repair, Enucleation, Excretion, Immune Response, Keratinization, Localization, Menopause, Methylation, Mismatch Repair, Oncogenesis, Pathogenesis, Pigmentation, Secretion

Quite a number of genes have been found to play important roles in Neoplasms, Multiple Primary, such as AFP, CALCA, CDKN2A, CEACAM5, CEACAM7, ENO2, KIT, KLK3, KRT20, KRT7, MLH1, MSH2, MUC1, POMC, PSG2, PTH, SLC25A5, TP53, VIM. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.

In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.