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Disease Info Card
Amputation Stumps
Information about Amputation Stumps: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Amputation Stumps
Most recent studies have shown that Amputation Stumps shares some biological mechanisms with amputation-traumatic, arm-injuries, arterial-occlusive-diseases, edema, finger-injuries, fracture, gangrene, hand-injuries, ischemia, leg-injuries, neoplasms, neuroma, pain, pain-in-limb, phantom-limb, surgical-wound-infection, ulcer, unspecified-disarticulation, vascular-diseases.
Among the many pathways, these few ones have gauged particular interests from scientists studying Amputation Stumps, and have been seen in publications frequently: Blood Circulation, Cell Proliferation, Coagulation, Dedifferentiation, Dehiscence, Dna Replication, Hypersensitivity, Innervation, Localization, Locomotion, Muscle Atrophy, Ossification, Pathogenesis, Reflex, Regeneration, Segmentation, Translation, Transport, Transposition, Wound Healing
Quite a number of genes have been found to play important roles in Amputation Stumps, such as ADK, CALM1, CALM2, CALM3, CAMKMT, CHRM1, GRIP1, HHIP, KRIT1, LIMS1, PMEL, PTBP1, PTBP2, PYCARD, REG3A, RPL29, SI, ST13, TYMS. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.