Disease Info Card

Agammaglobulinemia

Information about Agammaglobulinemia: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.

Overview of Agammaglobulinemia

Most recent studies have shown that Agammaglobulinemia shares some biological mechanisms with anemia, ataxia-telangiectasia, autoimmune-diseases, bacterial-infections, common-variable-immunodeficiency, dysgammaglobulinemia, hypogammaglobulinemia, immunoglobulin-a-deficiency-(disorder), immunologic-deficiency-syndromes, infective-disorder, leukemia, lymphoid-leukemia, lymphoma, neoplasms, pneumonia, primary-immune-deficiency-disorder, respiratory-tract-infections, thymoma, wiskott-aldrich-syndrome, x-linked-agammaglobulinemia.

Among the many pathways, these few ones have gauged particular interests from scientists studying Agammaglobulinemia, and have been seen in publications frequently: B Cell Differentiation, Cell Activation, Cell Development, Cell Differentiation, Cell Growth, Cell Maturation, Cell Proliferation, Chemotaxis, Complement Activation, Excretion, Humoral Immune Response, Hypersensitivity, Immune Response, Immunoglobulin Production, Localization, Lymphocyte Proliferation, Pathogenesis, Phagocytosis, Secretion, Transport

Quite a number of genes have been found to play important roles in Agammaglobulinemia, such as ADA, ALB, BTK, C3, CD19, CD4, CD40LG, CD8A, CTLA4, HLA-DQA1, IL2, IL6, NOD2, NT5E, TNFRSF13B, WAS. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.

In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.