Disease Info Card

Hypogammaglobulinemia

Information about Hypogammaglobulinemia: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.

Overview of Hypogammaglobulinemia

Most recent studies have shown that Hypogammaglobulinemia shares some biological mechanisms with agammaglobulinemia, anemia, autoimmune-diseases, autoimmune-reaction, bacterial-infections, chronic-lymphocytic-leukemia, common-variable-immunodeficiency, diarrhea, immunoglobulin-a-deficiency-(disorder), immunologic-deficiency-syndromes, infective-disorder, leukemia, lymphoid-leukemia, lymphoma, malignant-neoplasms, neoplasms, pneumonia, respiratory-tract-infections, thymoma, thymus-neoplasms.

Among the many pathways, these few ones have gauged particular interests from scientists studying Hypogammaglobulinemia, and have been seen in publications frequently: B Cell Differentiation, B Cell Proliferation, Cell Activation, Cell Development, Cell Differentiation, Cell Growth, Cell Proliferation, Chemotaxis, Coagulation, Complement Activation, Cytokine Production, Excretion, Hypersensitivity, Immune Response, Immunoglobulin Production, Immunoglobulin Secretion, Localization, Lymphocyte Proliferation, Pathogenesis, Secretion

Quite a number of genes have been found to play important roles in Hypogammaglobulinemia, such as ALB, BTK, C3, CD19, CD4, CD40LG, CD8A, CTLA4, CXCR4, HLA-DQA1, IL2, IL4, IL6, NOD2, NT5E, SH2D1A, TNF, TNFRSF13B. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.

In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.