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- Table of Contents
Information about Dysgammaglobulinemia: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Most recent studies have shown that Dysgammaglobulinemia shares some biological mechanisms with agammaglobulinemia, arthritis, asthma, ataxia-telangiectasia, autoimmune-diseases, autoimmune-reaction, bacterial-infections, dysglobulinemia, hypergammaglobulinemia, hypogammaglobulinemia, igg-deficiency, immunoglobulin-a-deficiency-(disorder), immunologic-deficiency-syndromes, infective-disorder, lupus-erythematosus-systemic, lymphoma, multiple-myeloma, neoplasms, pneumonia, respiratory-tract-infections.
Among the many pathways, these few ones have gauged particular interests from scientists studying Dysgammaglobulinemia, and have been seen in publications frequently: Anaphylaxis, B Cell Differentiation, Cell Activation, Cell Differentiation, Cell Proliferation, Chemotaxis, Coagulation, Humoral Immune Response, Hypersensitivity, Immune Complex Formation, Immune Response, Immunoglobulin Production, Lymphocyte Activation, Neutrophil Chemotaxis, Opsonization, Pathogenesis, Phagocytosis, Secretion, Sensitization, T Cell Activation
Quite a number of genes have been found to play important roles in Dysgammaglobulinemia, such as ALB, APCS, C2, C3, C4A, CD40LG, CD8A, CTLA4, HLA-A, HLA-DQA1, HNRNPC, IGHG3, IL2, IL4, LBR, NDUFA2, NOD2, SH2D1A, WAS. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.