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Disease Info Card
X-linked Agammaglobulinemia
Information about X-linked Agammaglobulinemia: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of X-linked Agammaglobulinemia
Most recent studies have shown that X-linked Agammaglobulinemia shares some biological mechanisms with agammaglobulinemia, arthritis, autoimmune-reaction, bacterial-infections, common-variable-immunodeficiency, congenital-combined-immunodeficiency, genetic-diseases-x-linked, granulomatous-disease-chronic, granulomatous-disorder, hyperimmunoglobulin-m-syndrome, hypogammaglobulinemia, immunoglobulin-a-deficiency-(disorder), immunologic-deficiency-syndromes, infective-disorder, lymphoma, malignant-neoplasms, pneumonia, primary-immune-deficiency-disorder, severe-combined-immunodeficiency, wiskott-aldrich-syndrome.
Among the many pathways, these few ones have gauged particular interests from scientists studying X-linked Agammaglobulinemia, and have been seen in publications frequently: B Cell Activation, B Cell Differentiation, Cell Activation, Cell Development, Cell Differentiation, Cell Maturation, Cell Proliferation, Cytokine Production, Excretion, Humoral Immune Response, Immune Response, Immunoglobulin Production, Isotype Switching, Localization, Lymphocyte Differentiation, Methylation, Pathogenesis, Phagocytosis, Secretion, Translation
Quite a number of genes have been found to play important roles in X-linked Agammaglobulinemia, such as BCR, BTK, CD19, CD40LG, CTLA4, HLA-DQA1, IL2, IL6, ITK, MS4A1, NOD2, PLEK, SRC, TNF, TNFRSF13B, WAS. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.