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Disease Info Card
Adams Oliver Syndrome
Information about Adams Oliver Syndrome: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Adams Oliver Syndrome
Most recent studies have shown that Adams Oliver Syndrome shares some biological mechanisms with aplasia-cutis-congenita, aplasia-nos, congenital-abnormality, congenital-absence, congenital-foot-deformity, congenital-hand-deformities, congenital-heart-defects, congenital-livedo-reticularis, cutis-marmorata, ectodermal-dysplasia, epilepsy, hemorrhage, hypoplasia, limb-defect, limb-deformities-congenital, nervousness, scalp-dermatoses, telangiectasis.
Among the many pathways, these few ones have gauged particular interests from scientists studying Adams Oliver Syndrome, and have been seen in publications frequently: Actin Cytoskeleton Organization, Brain Development, Cell Adhesion, Cell Proliferation, Cytokinesis, Cytoskeleton Organization, Fertilization, Limb Development, Ossification, Pathogenesis, Regeneration, Regulation Of Cell Proliferation, Vasculogenesis
Quite a number of genes have been found to play important roles in Adams Oliver Syndrome, such as ACACA, ALX1, ARHGAP31, BSND, CDC42, DOCK6, HES1, IGF1, MTSS1, MYH9, NF1, RAC1, TP63, TRAF4, UVRAG. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.