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Disease Info Card
Aplasia Cutis Congenita
Information about Aplasia Cutis Congenita: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Aplasia Cutis Congenita
Most recent studies have shown that Aplasia Cutis Congenita shares some biological mechanisms with adams-oliver-syndrome, aplasia-nos, atresia, cicatrix, congenital-abnormality, congenital-absence, dermatologic-disorders, diseases-in-twins, ectodermal-dysplasia, epidermolysis-bullosa, fetal-death, fetus-papyraceous, hemorrhage, limb-defect, limb-deformities-congenital, melanocytic-nevus, nervousness, scalp-dermatoses, skin-abnormalities.
Among the many pathways, these few ones have gauged particular interests from scientists studying Aplasia Cutis Congenita, and have been seen in publications frequently: Brain Development, Cell Adhesion, Cell Communication, Cell Migration, Cell Proliferation, Coagulation, Cytokinesis, Cytoskeleton Organization, Localization, Mating, Neural Tube Closure, Nipple Development, Ossification, Pathogenesis, Pigmentation, Regeneration, Skin Development, Tube Closure, Vasculogenesis, Wound Healing
Quite a number of genes have been found to play important roles in Aplasia Cutis Congenita, such as ACACA, ACHE, AFP, ARHGAP31, CALR, FN1, GNA11, LAMA3, LAMA4, LAMB3, LAMC2, NLRP5, RAC1, RICTOR, SSB, TLE2, UBR1. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.