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- Table of Contents
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pathway Info Card
Response To Methotrexate
Information about Response To Methotrexate: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Response To Methotrexate
Most recent studies have shown that Response To Methotrexate shares some biological mechanisms with cell-activation, cell-adhesion, cell-cycle, cell-proliferation, chemotaxis, complement-activation, drug-resistance, fertilization, g1-phase, hypersensitivity, inflammatory-response, menstruation, methotrexate-transport, neutrophil-chemotaxis, pathogenesis, secretion, secretory-pathway, t-cell-activation, translation, transport.
Among the many pathways, these few ones have gauged particular interests from scientists studying Response To Methotrexate, and have been seen in publications frequently: cell-activation, cell-adhesion, cell-cycle, cell-proliferation, chemotaxis, complement-activation, drug-resistance, fertilization, g1-phase, hypersensitivity, inflammatory-response, menstruation, methotrexate-transport, neutrophil-chemotaxis, pathogenesis, secretion, secretory-pathway, t-cell-activation, translation, transport
Quite a number of genes have been found to play important roles in Response To Methotrexate, such as ACR, ADA, ALB, ATIC, CD28, CD80, CRP, DHFR, ESR1, HLA-DRB1, IL10, IL6, ITPA, JAK3, MTHFR, MTX1, SLC19A1, TNF, TYMS. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this pathway. Plesae stay updated.