pathway Info Card

Endocardial Cushion Formation

Information about Endocardial Cushion Formation: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.

Overview of Endocardial Cushion Formation

Most recent studies have shown that Endocardial Cushion Formation shares some biological mechanisms with cell-activation, cell-adhesion, cell-development, cell-differentiation, cell-migration, cell-proliferation, developmental-process, embryo-development, fibroblast-proliferation, gastrulation, gene-silencing, heart-development, heart-looping, localization, muscle-cell-development, pathogenesis, transdifferentiation, transport, tube-formation, vascular-smooth-muscle-cell-development.

Among the many pathways, these few ones have gauged particular interests from scientists studying Endocardial Cushion Formation, and have been seen in publications frequently: cell-activation, cell-adhesion, cell-development, cell-differentiation, cell-migration, cell-proliferation, developmental-process, embryo-development, fibroblast-proliferation, gastrulation, gene-silencing, heart-development, heart-looping, localization, muscle-cell-development, pathogenesis, transdifferentiation, transport, tube-formation, vascular-smooth-muscle-cell-development

Quite a number of genes have been found to play important roles in Endocardial Cushion Formation, such as ACVR1, BMP2, BMP4, BMPR1A, FN1, HAS2, HBA1, ITK, KDR, PECAM1, PTPRJ, RBPJ, SLC22A3, SS18L1, TAL1, TBX2, TGFB1, VEGFA. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.

In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this pathway. Plesae stay updated.