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Disease Info Card
Endocardial Cushion Defects
Information about Endocardial Cushion Defects: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Endocardial Cushion Defects
Most recent studies have shown that Endocardial Cushion Defects shares some biological mechanisms with atresia, atrial-septal-defects, atrioventricular-septal-defect---isolated-atrial-component, common-atrioventricular-canal, congenital-abnormality, congenital-heart-defects, congenital-heart-disease, double-outlet-right-ventricle, down-syndrome, heart-diseases, heart-septal-defects, mitral-valve-insufficiency, patent-ductus-arteriosus, pulmonary-hypertension, regurgitation, stenosis, tetralogy-of-fallot, transposition-of-great-vessels, ventricular-septal-defects.
Among the many pathways, these few ones have gauged particular interests from scientists studying Endocardial Cushion Defects, and have been seen in publications frequently: Cardiac Chamber Formation, Cardiac Conduction, Cell Adhesion, Cell Migration, Cell Motility, Cell Proliferation, Dehiscence, Developmental Process, Endocardial Cushion Development, Endocardial Cushion Formation, Heart Development, Heart Looping, Heart Morphogenesis, Heart Valve Development, Heart Valve Formation, Localization, Mesenchymal Cell Migration, Pathogenesis, Secretion, Transposition
Quite a number of genes have been found to play important roles in Endocardial Cushion Defects, such as AMY2A, BLOC1S6, BMP2, BMP4, ECD, FN1, GATA4, GDF1, GJA1, GJA5, ITK, NKX2-5, PRH1, SLC22A3, SS18L1, TBX1, TBX2, TBX5. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.