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Disease Info Card
X-linked Dyskeratosis Congenita
Information about X-linked Dyskeratosis Congenita: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of X-linked Dyskeratosis Congenita
Most recent studies have shown that X-linked Dyskeratosis Congenita shares some biological mechanisms with anemia, aplastic-anemia, dermatologic-disorders, dyskeratosis, dyskeratosis-congenita, dystrophia-unguium, dystrophy, fetal-growth-retardation, keratosis, leukoplakia, leukoplakia-oral, malignant-neoplasms, microcephaly, multisystem-disorder, nails-malformed, neoplasms, pancytopenia, pigmentation-disorders.
Among the many pathways, these few ones have gauged particular interests from scientists studying X-linked Dyskeratosis Congenita, and have been seen in publications frequently: Aging, Cell Adhesion, Cell Cycle, Cell Cycle Checkpoint, Cell Division, Cell Proliferation, Cellular Senescence, Chromatin Remodeling, Localization, Oncogene-induced Senescence, Pathogenesis, Pigmentation, Ribosome Biogenesis, Rna Modification, Rna Processing, Rrna Processing, Senescence, Telomere Maintenance, Translation, Translational Initiation
Quite a number of genes have been found to play important roles in X-linked Dyskeratosis Congenita, such as APCDD1, DCX, DKC1, GALNT3, IFI27, MPP1, PSMD9, RNPC3, RUVBL2, SHQ1, TECR, TERT, TMED7, TNIP1, TP53, TRUB1, WRAP53, XIAP. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.