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Disease Info Card
Dystrophia Unguium
Information about Dystrophia Unguium: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Dystrophia Unguium
Most recent studies have shown that Dystrophia Unguium shares some biological mechanisms with bulla, dermatologic-disorders, dyskeratosis, dyskeratosis-congenita, dysplasia, dystrophy, ectodermal-dysplasia, epidermolysis-bullosa, hyperkeratosis, keratosis, leukoplakia, lichen-disease, nail-diseases, nails-malformed, onychomycosis, palmoplantar-keratosis, pancytopenia, psoriasis.
Among the many pathways, these few ones have gauged particular interests from scientists studying Dystrophia Unguium, and have been seen in publications frequently: Aging, Blood Circulation, Cell Adhesion, Coagulation, Cytolysis, Excretion, Exocytosis, Hypersensitivity, Immune Response, Keratinization, Keratinocyte Differentiation, Localization, Nail Development, Pathogenesis, Pigmentation, Sensitization, Sex Determination, Telomere Maintenance, Transport, Wound Healing
Quite a number of genes have been found to play important roles in Dystrophia Unguium, such as COL17A1, COL7A1, DKC1, DSP, DST, ENPP1, FOXN1, GJB2, GJB6, IKBKG, KRT14, KRT16, KRT17, KRT6A, MTSS1, PKP1, TPD52. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.