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Disease Info Card
Transfusion-related Acute Lung Injury
Information about Transfusion-related Acute Lung Injury: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Transfusion-related Acute Lung Injury
Most recent studies have shown that Transfusion-related Acute Lung Injury shares some biological mechanisms with acute-lung-injury, allergy, alloimmunization, blood-group-incompatibility, dyspnea, edema, fluid-overload, hemorrhage, hypotension-adverse-event, infective-disorder, lung-diseases, lung-injury, pulmonary-edema, respiratory-distress, respiratory-distress-syndrome-adult, systemic-infection, transfusion-reaction, transfusion-related-acute-lung-injury-(trali).
Among the many pathways, these few ones have gauged particular interests from scientists studying Transfusion-related Acute Lung Injury, and have been seen in publications frequently: Anaphylaxis, Cell Activation, Coagulation, Complement Activation, Diuresis, Donor Selection, Fibrinolysis, Hemostasis, Hypersensitivity, Immune Response, Inflammatory Response, Monocyte Activation, Neutrophil Activation, Pathogenesis, Phagocytosis, Platelet Activation, Respiratory Burst, Secretion, Sensitization, Vasoconstriction
Quite a number of genes have been found to play important roles in Transfusion-related Acute Lung Injury, such as ABO, ALB, ATR, AZU1, CD177, CD40LG, CORO1A, EPO, F2, FCGR2B, FCGR3A, FCGR3B, HLA-DRB4, ITGAL, ITGAM, NAAA, SHOX2, SLC44A2, TNF. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.