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Disease Info Card
Blood Group Incompatibility
Information about Blood Group Incompatibility: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Blood Group Incompatibility
Most recent studies have shown that Blood Group Incompatibility shares some biological mechanisms with alloimmunization, anemia, anemia-hemolytic, decreased-immunologic-activity-[pe], erythroblastosis-fetal, fetal-death, fetal-diseases, graft-vs-host-disease, haemolytic-transfusion-reaction, hemolysis-(disorder), hemolytic-disorder, hemorrhage, hyperbilirubinemia, icterus, neonatal-jaundice, pregnancy-complications, pregnancy-complications-hematologic, rh-isoimmunization, transfusion-reaction.
Among the many pathways, these few ones have gauged particular interests from scientists studying Blood Group Incompatibility, and have been seen in publications frequently: Anaphylaxis, Blood Coagulation, Coagulation, Complement Activation, Donor Selection, Excretion, Fibrinolysis, Glomerular Filtration, Hemostasis, Humoral Immune Response, Hypersensitivity, Immune Response, Inflammatory Response, Localization, Mating, Parturition, Pathogenesis, Regeneration, Sensitization, Tolerance Induction
Quite a number of genes have been found to play important roles in Blood Group Incompatibility, such as ABO, ALB, ATP6V0A1, ATP6V0A2, EPO, G6PD, GPHA2, HLA-DRB4, KRT20, MS4A1, RFC1, RFC2, RFC4, RHCE, RHD, SGCA, SLC6A3, TNF. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.